In the absence of the protein biglycan, synapses at neuromuscular junctions in mice began to break up about five weeks after birth, according to a new study. Reintroducing byglycan helped fix the loss of synaptic stability in cell culture. The research may be relevant to efforts to treat motor neuron diseases, such as amyotrophic lateral sclerosis (ALS, Lou Gherig’s Disease) and spinal muscular atrophy.
Quoted from When nerve meets muscle, biglycan seals the deal on ScienceDaily: Top News
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